mm.rcast u-tokyo.ac.jp

Home 東京大学 代謝医学分野 酒井研究室

WELCOME TO SAKAI LAB. ヒストンのメチル化などを解する エピジェネティックな遺伝子発現の制御 後天的遺伝子修飾 が細胞分裂を越えて保存され一個体の間の細胞記憶システムを形成しこのことが生活習慣病の 発症に大きな役割を果たしていることが明らかにされつつあります 祝 大口裕人 先生 酒井研OB が熊本大学 に独立准教授としてご就任. エコEko Fuji Ariyantoさん 学位取得. Overexpression of p54nrbNONO induces differential EPHA6 splicing and contributes to castration-resistant prostate cancer growth. Degradation of human Lipin-1 by BTRC E3 ubiquitin ligase. KDM3A coordinates actin dynamics with intraflagellar transport to regulate cilia stability.
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TITLE

Home 東京大学 代謝医学分野 酒井研究室

DESCRIPTION

WELCOME TO SAKAI LAB. ヒストンのメチル化などを解する エピジェネティックな遺伝子発現の制御 後天的遺伝子修飾 が細胞分裂を越えて保存され一個体の間の細胞記憶システムを形成しこのことが生活習慣病の 発症に大きな役割を果たしていることが明らかにされつつあります 祝 大口裕人 先生 酒井研OB が熊本大学 に独立准教授としてご就任. エコEko Fuji Ariyantoさん 学位取得. Overexpression of p54nrbNONO induces differential EPHA6 splicing and contributes to castration-resistant prostate cancer growth. Degradation of human Lipin-1 by BTRC E3 ubiquitin ligase. KDM3A coordinates actin dynamics with intraflagellar transport to regulate cilia stability.

CONTENT

This website has the following on the site, "ヒストンのメチル化などを解する エピジェネティックな遺伝子発現の制御 後天的遺伝子修飾 が細胞分裂を越えて保存され一個体の間の細胞記憶システムを形成しこのことが生活習慣病の 発症に大きな役割を果たしていることが明らかにされつつあります 祝 大口裕人 先生 酒井研OB が熊本大学 に独立准教授としてご就任." Our analyzers viewed that the web page also said " Overexpression of p54nrbNONO induces differential EPHA6 splicing and contributes to castration-resistant prostate cancer growth." The Website also said " Degradation of human Lipin-1 by BTRC E3 ubiquitin ligase. KDM3A coordinates actin dynamics with intraflagellar transport to regulate cilia stability."

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